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Collectively, the failure of these clinical studies has raised doubts about the HDL hypothesis

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2021;8 doi: 10.3389/fcvm.2021.707138

In the same context, in vitro studies suggest that SOCS3 directly interacts with the INSR, inhibiting IRS1 tyrosine phosphorylation and decreasing insulin-stimulated glycogen synthesis in cultured myotubes [58]